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Re: Heredity B/Designer Genes C

Posted: November 23rd, 2018, 10:48 am
by jxxu20
1. Klinefelter Syndrome
2. Turner Syndrome
3. Down Syndrome

Re: Heredity B/Designer Genes C

Posted: November 24th, 2018, 7:50 am
by Tailsfan101
jxxu20 wrote:1. Klinefelter Syndrome
2. Turner Syndrome
3. Down Syndrome
Correct, your turn. Also, remember to use
 when you post your answers.

Re: Heredity B/Designer Genes C

Posted: November 26th, 2018, 4:10 pm
by jxxu20
Whoops.

1. In poultry, the dominant allele B can result in black feathers. Its other allele, b, can produce white splashed feathers. The factors O and C are both required for any color at all in the feathers. Homozygous recessiveness at either locus results in white feathers. Two completely white birds mate and produce an F1 that is all black. What are the genotypes of the parents and the progeny?

2. How is pre-mRNA processed?

3. How does cytokinesis occur in plant cell mitosis, and how is this different from cytokinesis in animal cells?

4. What is the Shine-Dalgarno sequence and its function?

Re: Heredity B/Designer Genes C

Posted: December 19th, 2018, 4:24 pm
by Tailsfan101
Since this seems to have died, I'll post some new questions.

For questions 1-10, list whether the disorder is:
a. autosomal dominant
b. autosomal recessive
c. X-linked dominant
d. X-linked recessive
e. Y-linked
f. nondisjunction

1. hairy ears
2. spina bifida
3. Beta thalassemia
4. Tay-Sachs Disease
5. Nonsyndromic deafness
6. Chronic simple glaucoma
7. ichthyosis simplex
8. Lesch-Nyhan Disease
9. Phenylketonuria
10. Fragile X Syndrome

Re: Heredity B/Designer Genes C

Posted: December 25th, 2018, 9:56 am
by SonicSpeed
Tailsfan101 wrote:Since this seems to have died, I'll post some new questions.

For questions 1-10, list whether the disorder is:
a. autosomal dominant
b. autosomal recessive
c. X-linked dominant
d. X-linked recessive
e. Y-linked
f. nondisjunction

1. hairy ears
2. spina bifida
3. Beta thalassemia
4. Tay-Sachs Disease
5. Nonsyndromic deafness
6. Chronic simple glaucoma
7. ichthyosis simplex
8. Lesch-Nyhan Disease
9. Phenylketonuria
10. Fragile X Syndrome
1. e
2. Isn't it multifactorial?
3. b
4. b
5. a
6. a
7. c
8. d
9. b
10. d

Re: Heredity B/Designer Genes C

Posted: December 26th, 2018, 10:19 am
by Tailsfan101
SonicSpeed wrote:
Tailsfan101 wrote:Since this seems to have died, I'll post some new questions.

For questions 1-10, list whether the disorder is:
a. autosomal dominant
b. autosomal recessive
c. X-linked dominant
d. X-linked recessive
e. Y-linked
f. nondisjunction

1. hairy ears
2. spina bifida
3. Beta thalassemia
4. Tay-Sachs Disease
5. Nonsyndromic deafness
6. Chronic simple glaucoma
7. ichthyosis simplex
8. Lesch-Nyhan Disease
9. Phenylketonuria
10. Fragile X Syndrome
1. e
2. Isn't it multifactorial?
3. b
4. b
5. a
6. a
7. c
8. d
9. b
10. d
Oh, whoops, I forgot to put multifactorial on there! All correct, your turn.

Re: Heredity B/Designer Genes C

Posted: December 27th, 2018, 5:58 pm
by platypusomelette
jxxu20 wrote:Whoops.

1. In poultry, the dominant allele B can result in black feathers. Its other allele, b, can produce white splashed feathers. The factors O and C are both required for any color at all in the feathers. Homozygous recessiveness at either locus results in white feathers. Two completely white birds mate and produce an F1 that is all black. What are the genotypes of the parents and the progeny?

2. How is pre-mRNA processed?

3. How does cytokinesis occur in plant cell mitosis, and how is this different from cytokinesis in animal cells?

4. What is the Shine-Dalgarno sequence and its function?
1. No idea
2. Immediately after transcription is terminated, a poly-A tail is added to the 3' end and a modified guanine cap is added to the 5' end. Complexes called spliceosomes (made of snRNPs, which are complexes of protein and small nuclear RNA) recognize and splice intron sites, leaving behind only exons, which are later expressed as protein.
3. The cell is split by the growth of a cell plate, which is assembled by vesicles from the golgi body, and cytokinesis in animal cells is carried out by an actin filament ring that pinches the cell in half. I don't know the details of this so if you have a full answer pls tell lol
4. I'm not sure about this one either I think it's the sequence that the ribosome initially sticks to in the beginning of translation in prokaryotes?

Re: Heredity B/Designer Genes C

Posted: December 27th, 2018, 6:06 pm
by platypusomelette
1. What is the difference between a transversion and a transition?
2. Explain how RFLPs can be used to identify an individual.
3. What kinds of nucleotides are used in sanger sequencing?
4. What is the purpose of eukaryotic control elements?
5. Name some examples of prezygotic and postzygotic barriers.

Re: Heredity B/Designer Genes C

Posted: December 28th, 2018, 11:43 am
by jxxu20
@platypusomelette Good job!

1: The factors O and C are epistatic genes (they cause the bird to be white), and the parents are both completely white. However, their children (progeny) all have black feathers. Hence, both parents must have a BB genotype for the color gene. Going back to the epistatic gene, homozygous recessiveness for either will result in white coloration, which is present in the parents. However, since the children are colored we know that the parents have a homozygous recessive genotype for one epistatic gene (either O or C), and they must be homozygous dominant at the other locus (since the children are all colored). This must mean that the genotypes of the parents are BBOOcc and BBooCC. Their children will have a genotype of BBOoCc.

3. That's what I have in my notes -- sorry xD

4. Yup! It's usually located somewhere 8-10 bases upstream of the start codon AUG and helps recruit the ribosome for translation.

Re: Heredity B/Designer Genes C

Posted: January 5th, 2019, 6:08 pm
by platypusomelette
Bumpo