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Actually, one of my textbooks also says that salivary lipase exists. Whether or not it actually begins breaking down fats in the mouth, though, is not in the text.

foreverphysics wrote:

mnstrviola wrote:

foreverphysics wrote:I don't think the information is really that debatable. I looked in my textbook today (it has a table of every chemical and where it is secreted), and nowhere in the "saliva" section did it say "lipase". The only chemical that saliva had listed under it was amylase. All the lipase stuff was listed under pancreas. So...either your Anatomy book is wacked up, or mine is.

I'm 99.9999...% sure that salivary lipase at least exists.

If it does, it's not mentioned in my textbook. Make of that what you will.

My textbook says salivary lipase exists...

For division b, we don't have to know any formulas (aka snell's law, thin lens equation, lens makers equation, ect.) right???

geekychic13 wrote:For division b, we don't have to know any formulas (aka snell's law, thin lens equation, lens makers equation, ect.) right???

Was this supposed to be in the Optics thread?

geekychic13 wrote:For division b, we don't have to know any formulas (aka snell's law, thin lens equation, lens makers equation, ect.) right???

Just to stop this from going any further, you need to know ALL those things. And more. It's not that hard, trust me. And I suggest next time actually posting in the right place. Thank me on the Optics thread and not the Anatomy one.

Here's another question for you guys...

What would be the effect of viral pneumonia on the lung capacities? Such as tidal volume, expiratory reserve volume, and total lung capacity?

kappakun wrote:Here's another question for you guys...

What would be the effect of viral pneumonia on the lung capacities? Such as tidal volume, expiratory reserve volume, and total lung capacity?

Pneumonia is inflammation of mostly alveoli. I don't think the inflammation actually increases the usable volume of the alveoli but instead makes it more difficult to breathe. Therefore, TV and total lung capacity should probably decrease, not sure about ERV.

Hey, for you people who were wondering about the salivary lipase thing and the breakdown of lipids in the mouth, I just confirmed that salivary lipase is activated in the stomach because salivary pH does not reach optimum acidity levels for salivary lipase to actually do anything. Chemical digestion in the mouth is solely restricted to carbohydrates.

This is what my book says....
"...intrinsic salivary glands contain lingual lipase, a fat-digesting enzyme that is optimally active at an acid pH."
Later whie discussing the digestion of lipids it says:
"The small intestine is essentially the sole site of lipid digestion because the pancreas is the only significant source of fat-digesting enzymes, or lipases"
Since lingual lipase is optimally active at a low pH,it seems logocal that is must work in the acidic stomach, the amount of digestion it causes must be miniscule though, if essentailly all fat digestion occurs in the pancreas. Lingual lipase might not be mentioned in some textbooks b/c it causes such a small amount of digeston.

rkoopma2 wrote:This is what my book says....
"...intrinsic salivary glands contain lingual lipase, a fat-digesting enzyme that is optimally active at an acid pH."
Later whie discussing the digestion of lipids it says:
"The small intestine is essentially the sole site of lipid digestion because the pancreas is the only significant source of fat-digesting enzymes, or lipases"
Since lingual lipase is optimally active at a low pH,it seems logocal that is must work in the acidic stomach, the amount of digestion it causes must be miniscule though, if essentailly all fat digestion occurs in the pancreas. Lingual lipase might not be mentioned in some textbooks b/c it causes such a small amount of digeston.

***Imaginary High Five***